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ERK 1/2 activation does not contribute to increased survival mediated by bone marrow cells after 90% partial hepatectomy in wistar rats

dc.contributor.authorMuñoz, Gustavo Alfredo Ochs dept_BR
dc.contributor.authorKretzmann, Nelson Alexandrept_BR
dc.contributor.authorLópez, Mónica Lujánpt_BR
dc.contributor.authorKieling, Carlos Oscarpt_BR
dc.contributor.authorCruz, Carolina Uribept_BR
dc.contributor.authorSilva, Luciano Basso dapt_BR
dc.contributor.authorOsvaldt, Alessandro Berschpt_BR
dc.contributor.authorMatte, Ursula da Silveirapt_BR
dc.date.accessioned2017-05-30T02:37:19Zpt_BR
dc.date.issued2016pt_BR
dc.identifier.issn2357-9730pt_BR
dc.identifier.urihttp://hdl.handle.net/10183/158741pt_BR
dc.description.abstractWe investigated the influence of bone marrow cells upon activation of ERK ½ in an animal model of 90% PH. Phosphorylated ERK 1/2 was evaluated by western blot. No differences were found between the groups. Thus, increased survival does not appear to be mediated by ERK 1/2 activation.en
dc.format.mimetypeapplication/pdfpt_BR
dc.language.isoengpt_BR
dc.relation.ispartofClinical and biomedical research. Porto Alegre. Vol. 36, n. 4, (2016), p. 254-256pt_BR
dc.rightsOpen Accessen
dc.subjectMAP Kinase Signaling Systemen
dc.subjectMAP quinases reguladas por sinal extracelularpt_BR
dc.subjectCélulas-troncopt_BR
dc.subjectStem cellsen
dc.subjectInsuficiência hepáticapt_BR
dc.subjectAcute liver failureen
dc.titleERK 1/2 activation does not contribute to increased survival mediated by bone marrow cells after 90% partial hepatectomy in wistar ratspt_BR
dc.typeArtigo de periódicopt_BR
dc.identifier.nrb001012859pt_BR
dc.type.originNacionalpt_BR


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